Achilles Tendon by Andrej Čretnik

By Andrej Čretnik

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British Journal of Sports Medicine, 44(16), 1160–1165. Posthumus M, Collins M, Cook J, Handley CJ, Ribbans WJ, Smith RK, Schwellnus MP, and Raleigh SM. (2010). Components of the transforming growth factor-beta family and the pathogenesis of human Achilles tendon pathology--a genetic association study. Rheumatology (Oxford) 49: 2090-2097. , & Postacchini, F. (1976). A classification of Achilles tendon disease. The American Journal of Sports Medicine, 4(4), 145–150. Raleigh SM, van der Merwe L, Ribbans WJ, Smith RK, Schwellnus MP, and Collins M.

These possible mechanism need to be proved or verified using other biological techniques. As previously reviewed (Collins 34 Achilles Tendon & Posthumus, In Press) work is currently ongoing to explain how variants within the 3’UTR of the COL5A1 gene could be directly involved in the susceptibility to Achilles tendon injuries. One of the reasons the current risk models (figure 1) have limited practical application in determining risk of injury for a individual athlete is that they are not based on an understanding on the biological mechanisms causing the injury.

1998). , 1997). The diversity of the cytokinetic characteristics lends credence to the concept that these growth factors are potentially capable of modulating the tendon healing processes, resulting in better healing (Mason & Allen 1941). The influence of growth factors on tendon healing has been investigated through comparative studies of the profiles of cytokines of injured and normal tendons. , 1998; Party et al 1978). , 1998). Under the influence of PDGF, rates of fibroblasts proliferation, chemotaxis and collagen synthesis are somewhat elevated (O'Brien T, 1992).

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